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Patobiologi Molekuler Kanker

Malignancy (cancer) is one of the diseases that cannot be handled optimally. Along with the development of technology, within a certain period of time, the disease is expected to be handled optimally. To study the development of cancer control technology, an understanding of cancer pathobiology seems to be very necessary both at the molecular and cellular levels. It is known that the transformation of cells towards malignancy is triggered by a mutagen that causes the DNA of the cell to experience a mutation, namely the accumulation of mutations (Multi Gene Defect), especially genes that regulate the cell division cycle. Physiologically, cells have a very tolerant nature to their environment, where cells always maintain their uniformity and function in accordance with their existence. Therefore, if the cell experiences a gene defect, then with a certain mechanism it tries to repair it (gene repair mechanism). However, if the gene repair ability fails, the cell will try to execute itself through a mechanism known as autoapoptotic. Likewise, if the autoapoptotic ability fails, the cell attempts to express foreign proteins on its surface, so that the immune system recognizes the cell with the gene defect as a foreign cell (non-self).

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Informasi Detail
Judul Seri
Patobiologi Molekuler Kanker
No. Panggil
616.99 IKE p
Penerbit
Jakarta : Salemba Medika.,
Deskripsi Fisik
vi, 104 halaman : ilustrasi ; 21 cm.
Bahasa
Indonesia
ISBN/ISSN
978-979-3027-73--9
Klasifikasi
616.99
Tipe Isi
text
Tipe Media
unmediated
Tipe Pembawa
volume
Edisi
-
Subjek
Neoplasms/ pathology
Info Detail Spesifik
-
Pernyataan Tanggungjawab
Versi lain/terkait

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